Interleukin-6 as an Oral-Vascular Inflammatory Bridge: Integrating CXCL10 and Haptoglobin 2-2 into a Potential Model of Periodontitis-Associated Cardiovascular Risk.
Researchers propose that interleukin-6, together with the chemokine CXCL10 and the haptoglobin 2-2 phenotype, may explain how chronic periodontitis contributes to cardiovascular disease risk through systemic inflammation and vascular dysfunction.
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